A new study published in Experimental Gerontology found that among 170 blood donors, elderly patients with heart failure had the lowest NAD+ levels, which suggests a role for NAD+ precursors in supporting heart health [1].
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A recent study found that raising NAD+ levels in a mouse model could protect the survival and function of retinal cells, the cells responsible for sight, against damaging UV light.
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Raising NAD+ levels with the precursor Nicotinamide Riboside (NR) was found to reduce markers of inflammation in the blood from a small group of Stage D heart failure (HF) patients.
A new study shows that while both nicotinamide mononucleotide (NMN) and nicotinamide riboside (NR) can help protect against blood-vessel inflammation by raising NAD+, NR may be the more direct and effective precursor.
The latest research suggests NAD+ may play a central role in regulating inflammation for an effective—but targeted—immune response to dangerous pathogens such as the Coronavirus.
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The human body is composed of cells in the trillions. Each cell relies on a resource molecule called NAD+ for the generation of energy we know as ATP. This energy helps cells perform their unique functions and survive. As the available knowledge of NAD+ expands, scientists have come to realize that it also plays a vital role in helping activate important signaling molecules for cellular defense.
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Just like our planet is mustering up the resources to fight the ongoing COVID-19 (Coronavirus) pandemic, so too do our cells need energy to support cellular repair. NAD+ may hold the key to supporting cellular resilience in the face of immune stress, like viral infections.
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